yeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just trenholy overcomplicating ts just blast the 5 good roids and u mog
literally yesyeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just tren
Thoughts on Eli lilly's myostatin inhibitors? Bimagrumab, Domagrozumab etc?Base : Test
Genomic pathways : Tren , Ment
Non genomic pathways : Anadrol
Other pathways : HGH + Slin (not rlly AAS) , GCR agonists , YK-11 (Myostatin inhibition) , β2-adrenergic pathway (Clenbuterol)
You can't make huge blanket statements though many serve different purposes and have situational use
Don't even bother this nigga is blasting 700mg of tren no ancillaries for what i know.yeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just tren
Don't even bother this nigga is blasting 700mg of tren no ancillaries for what i know.
nah not me broThoughts on Eli lilly's myostatin inhibitors? Bimagrumab, Domagrozumab etc?
now imagine the gains you would've made if you did a molecule of researchliterally yes
View attachment 55204
Selectivity tho....for what i've read they bind a fuck ton to activin.
imagine the gains u would have made if u actually did something instead of js larping roidingnow imagine the gains you would've made if you did a molecule of research
cringe gymbro mesorx tier userimagine the gains u would have made if u actually did something instead of js larping roidingView attachment 55206
Though it looks like we can't expect the results obtained in animal studies, the look of dogs and rats with myostatin deficiencies that looked insane asf where not observed in human trials, the human body seems to perform exceptionally well when it comes to maintaining homeostasis
mesorx unc is dream build bro whatcringe gymbro mesorx tier user
main issues caused by that you're getting roids alreadySelectivity tho....for what i've read they bind a fuck ton to activin.
the trial i referenced was a human trialThough it looks like we can't expect the results obtained in animal studies, the look of dogs and rats with myostatin deficiencies that looked insane asf where not observed in human trials, the human body seems to perform exceptionally well when it comes to maintaining homeostasis
I think i phrased it poorly, what i meant was that the results regarding muscle growth on animal vs human trials were super different, for what I've seen humans didn't react like rats or cows (that got insanely muscular) eg;the trial i referenced was a human trial


