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Discussion What are the best aas??? (24 Viewers)

Discussion What are the best aas???

Benji184

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  • #1
Caption
 
Solution
Base : Test
Genomic pathways : Tren , Ment
Non genomic pathways : Anadrol
Other pathways : HGH + Slin (not rlly AAS) , GCR agonists , YK-11 (Myostatin inhibition) , β2-adrenergic pathway (Clenbuterol)

You can't make huge blanket statements though many serve different purposes and have situational use

surgerymax

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  • #16
holy overcomplicating ts just blast the 5 good roids and u mog
yeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just tren
 

urelos

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  • #17
yeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just tren
literally yes
AB94F2EA-E6EF-4883-A892-BAB9866752CF.jpeg
 

Daker

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  • #18
Base : Test
Genomic pathways : Tren , Ment
Non genomic pathways : Anadrol
Other pathways : HGH + Slin (not rlly AAS) , GCR agonists , YK-11 (Myostatin inhibition) , β2-adrenergic pathway (Clenbuterol)

You can't make huge blanket statements though many serve different purposes and have situational use
Thoughts on Eli lilly's myostatin inhibitors? Bimagrumab, Domagrozumab etc?
 

Daker

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  • #19
yeah bro blast 5 roids all hitting the same pathway thats already saturated to death by just tren
Don't even bother this nigga is blasting 700mg of tren no ancillaries for what i know.
 

urelos

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  • #20

surgerymax

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  • #21
Thoughts on Eli lilly's myostatin inhibitors? Bimagrumab, Domagrozumab etc?
1781049225459.png


domagrozumab is pretty shit but bimagrumab seems cracked
 

surgerymax

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  • #22

Daker

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  • #23

urelos

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  • #24
now imagine the gains you would've made if you did a molecule of research
imagine the gains u would have made if u actually did something instead of js larping roiding
5BFC4C00-100E-4850-BF68-E11528AB9591.png
 

surgerymax

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  • #25

Daker

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  • #26
View attachment 55205

domagrozumab is pretty shit but bimagrumab seems cracked
Though it looks like we can't expect the results obtained in animal studies, the look of dogs and rats with myostatin deficiencies that looked insane asf where not observed in human trials, the human body seems to perform exceptionally well when it comes to maintaining homeostasis
 

urelos

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  • #27

surgerymax

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  • #28
Selectivity tho....for what i've read they bind a fuck ton to activin.
main issues caused by that you're getting roids already
- already suppressed HPG axis
- already elevated HCT
- cardiac effects
- bone metabolism offset by androgens prob
good ancillary stack will already take care of these issues
 

surgerymax

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  • #29
Though it looks like we can't expect the results obtained in animal studies, the look of dogs and rats with myostatin deficiencies that looked insane asf where not observed in human trials, the human body seems to perform exceptionally well when it comes to maintaining homeostasis
the trial i referenced was a human trial
 

Daker

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  • #30
the trial i referenced was a human trial
I think i phrased it poorly, what i meant was that the results regarding muscle growth on animal vs human trials were super different, for what I've seen humans didn't react like rats or cows (that got insanely muscular) eg;

1781050091406.png

Brutal mog LTM vs HTM (low tier mouse vs high tier mouse)

Though I'm not sure if this pics are from rats on myostatin inhibitors or already genetically modified.
 

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