Full Androgen Support guide
by:
TheCoolDude2000
When you want to hop on roids you want to look like this:
And absolutely not like this:
And how do you stay healthy, maintain fertility and minimize damage your organs and cardiovascular health?
So how do we prevent/manage the undesired side effects from androgen use?
First we need to look at all the potential side effects and what compounds and dosages give certain sides. The following study proves this claim. https://www.researchgate.net/public...eroid_administration_in_male_normal_volunteer
So now we know this its ofc very difficult to know how you will respond. I will go over a few commonly used artillery's for AAS and how they work + doses and any other information.
Aromatise Inhibitors
Maybe the most know thing people take on AAS. Armotase Inhibitors are compounds that reduce the conversion between androgens into estrogens by simply inhibiting the aromatase enzyme. For males means that testosterone is being converted into estradiol. Estradiol, also known as E2 isnt a bad hormone but has variys functions in the body and a stable level is very healthy for things like BMD, brain/mood and Libido/Sexual Function. Stable levels are around 10-20 ng/dl. But if you take exogenous testosterone or other aromatizing AAS the level may become too high and lead too: gyno, early growth plate closure, bloating, libido problems and more.
Aromasin
Aromasin, or exemestane, is a steroidal aromatase inhibitor that binds irreversibly to aromatase and permanently deactivates it, so the body must produce new enzyme to restore estrogen production.
commun doses:
6,25mg = ~30-35% E2 reduction
12.5 mg = 55-60% E2 reduction
Arimidex
Arimidex, whose name is anastrozole, is a non-steroidal aromatase inhibitor that binds reversibly to the enzyme, meaning its effect depends on continued presence in the body. So it doesn't disable the enzyme
commun doses:
0.5mg = ~50% E2 suppression
1mg = ~80% E2 suppression
There are more inhibitors but these i would recommend when using an aromatizing compound like testosterone. Those numbers given are averages. I have stated earlier on that response to gear and his side effects variy by person. So the safest way to getting your dose right is with a pre-cycle bloodwork and a cycle bloodwork a few weeks in to adjust your dose around the 10-20 pg/dl.
Prolactin issues
Aromatizable steroids, such as testosterone, can increase estradiol levels through aromatization. Elevated estradiol can stimulate prolactin secretion like be handled before so keeping your E2 in check will help.
19-nor compounds like nandrolone and trenbolone are often associated with prolactin-related side effects, but evidence suggests they do not directly raise prolactin in most users. Instead, they enhance progesterone receptor activity, this can increase sensitivity of breast tissue and contribute to symptoms such as gyno or erectil dysfunction. In some individuals, prolactin may rise modestly, while in others it remains normal, showing variability in response.
P5P (pyridoxal-5-phosphate)
is the active form of vitamin B6 and plays a role in dopamine synthesis, which in turn inhibits prolactin release from the pituitary. Because prolactin is largely controlled by dopamine, supporting dopamine production with adequate B6/P5P may help mildly elevated prolactin levels in some individuals.
Cabergoline
It is dopamine antagonist that directly stimulates dopamine D2 receptors the pituitary. Cabergoline is much more potent than P5P and can lower prolactin rapidly and substantially, but it also carries potential side effects such as nausea, dizziness, low blood pressure, and, with long-term high-dose use, rare cardiac valve concerns.. Caber is way more potent and for most users is p5p dosing around 100-200mg enough on cycle. I would only recommend if bloodwork show still very high levels of prolactin even after p5p use.
Acne
Acne is a common side effect of anabolic-androgenic steroids because AAS increase androgen activity in the skin, which stimulates sebaceous glands to produce more oil. Higher sebum production, combined with changes in skin cell turnover and bacterial growth, can clog pores and lead to inflammatory acne. Ofc this also is really depending on how you respond.
Isotretinoin (Accutune)
Isotretinoin dramatically reduces the sebum production making it very effective for ‘roid acne’. Its unfortunate side effect is the significant stress on the liver it causes. Even if you already use compounds that have a negative impact on the liver like orals. Also it can have a positive influence on anti aging.
Liver protection
When people talk about liver stress from anabolic-androgenic steroids, they’re usually talking about oral compounds like methandrostenolone (Dianabol), oxandrolone (Anavar), stanozolol (Winstrol), oxymetholone (Anadrol) and Isotretinoin (Accutune) we mentioned before. The big issue is the 17-alpha-alkylated structure used in many oral AAS. With repeated exposure, this can elevate liver enzymes such as ALT and AST, indicating hepatocellular stress.
NAC
Nac also know as N-acetylcysteine is a commonly used as a supportive compound for the liver. NAC is a precursor to glutathione, one of the body’s main intracellular antioxidants. Glutathione helps neutralize reactive oxygen species and supports detoxification pathways in the liver helping it elp reduce oxidative stress and support liver enzyme. !!Note that this a supportive drug and not protective, that means it cant save your liver from 4 different stupidly doses orals.!!
Commun doses are around 600-1800mg depending on compounds and doses.
LDL/HDL issues
Some AAS may severely worsen cholesterol markes, by increasing LDL and suppressing HDL. LDL is also know as the ‘bad’ cholesterol and HDL as the ‘good’ one that clears LDL out. AAS use tends to tilt this scale aggressively in the wrong direction. Not what we want here comes:
Ezetimibe:
Ezetimibe reduces intestinal cholesterol absorption, which can help help with limiting LDL elevation. LDL Reduction: Typically reduces LDL by 15–22% when used alone. Its has only a very mild effect on HDL only increasing it by a few %.
Common used dosages are 10mg ed
Hairloss
The use of AAS turn you into to a bald chud gymcel, we for sure dont want this to happen. But how does it work? It really is depending on the compound. Hairloss mainly accors from DHT in males. DHT is very potent and has a high affinity for receptors in the scalp.
Once DHT binds to these receptors, it triggers a process called follicle miniaturization. The growth phase of the hair becomes shorter, the hair shaft becomes thinner, and eventually, the follicle stops producing hair entirely and falls out. Hairloss is mainly genetic and ofc it really matters if your hair is sensitive or not. Roiding can heavily speed up this process.
Finasteride and Dutasteride
There job is to inhibit the 5-alpha reductase (5-AR) enzyme, which is responsible for converting Testosterone into DHT. Inhibiting this could result in very low to non DHT what has some potential side effects like lower libido and mood. This is a very effective solution but is only interesting if you only take test. DHT derivatives are already structurally similar to DHT, the 5-AR enzyme has nothing to do with them. And 19-nor derivatives have zero influence on the 5-AE enzyme and taking Nandrolone can make it worse. Also wait until you are finished with puberty with taking this since DHT plays a big role in development.
RU58841
The magical solution if your not only taking test?
How does it work? Ru58841 is a topical androgen receptor antagonist used to local block the androgen activity at the hair follicle, helping with local blocking except eliminating DHT systemly and is very good if you take DHT derivatives or things like 19-nor derivatives.
by:
TheCoolDude2000 When you want to hop on roids you want to look like this:
And absolutely not like this:
And how do you stay healthy, maintain fertility and minimize damage your organs and cardiovascular health?
So how do we prevent/manage the undesired side effects from androgen use?
First we need to look at all the potential side effects and what compounds and dosages give certain sides. The following study proves this claim. https://www.researchgate.net/public...eroid_administration_in_male_normal_volunteer
So now we know this its ofc very difficult to know how you will respond. I will go over a few commonly used artillery's for AAS and how they work + doses and any other information.
Aromatise Inhibitors
Maybe the most know thing people take on AAS. Armotase Inhibitors are compounds that reduce the conversion between androgens into estrogens by simply inhibiting the aromatase enzyme. For males means that testosterone is being converted into estradiol. Estradiol, also known as E2 isnt a bad hormone but has variys functions in the body and a stable level is very healthy for things like BMD, brain/mood and Libido/Sexual Function. Stable levels are around 10-20 ng/dl. But if you take exogenous testosterone or other aromatizing AAS the level may become too high and lead too: gyno, early growth plate closure, bloating, libido problems and more.
Aromasin
Aromasin, or exemestane, is a steroidal aromatase inhibitor that binds irreversibly to aromatase and permanently deactivates it, so the body must produce new enzyme to restore estrogen production.
commun doses:
6,25mg = ~30-35% E2 reduction
12.5 mg = 55-60% E2 reduction
Arimidex
Arimidex, whose name is anastrozole, is a non-steroidal aromatase inhibitor that binds reversibly to the enzyme, meaning its effect depends on continued presence in the body. So it doesn't disable the enzyme
commun doses:
0.5mg = ~50% E2 suppression
1mg = ~80% E2 suppression
There are more inhibitors but these i would recommend when using an aromatizing compound like testosterone. Those numbers given are averages. I have stated earlier on that response to gear and his side effects variy by person. So the safest way to getting your dose right is with a pre-cycle bloodwork and a cycle bloodwork a few weeks in to adjust your dose around the 10-20 pg/dl.
Prolactin issues
Aromatizable steroids, such as testosterone, can increase estradiol levels through aromatization. Elevated estradiol can stimulate prolactin secretion like be handled before so keeping your E2 in check will help.
19-nor compounds like nandrolone and trenbolone are often associated with prolactin-related side effects, but evidence suggests they do not directly raise prolactin in most users. Instead, they enhance progesterone receptor activity, this can increase sensitivity of breast tissue and contribute to symptoms such as gyno or erectil dysfunction. In some individuals, prolactin may rise modestly, while in others it remains normal, showing variability in response.
P5P (pyridoxal-5-phosphate)
is the active form of vitamin B6 and plays a role in dopamine synthesis, which in turn inhibits prolactin release from the pituitary. Because prolactin is largely controlled by dopamine, supporting dopamine production with adequate B6/P5P may help mildly elevated prolactin levels in some individuals.
Cabergoline
It is dopamine antagonist that directly stimulates dopamine D2 receptors the pituitary. Cabergoline is much more potent than P5P and can lower prolactin rapidly and substantially, but it also carries potential side effects such as nausea, dizziness, low blood pressure, and, with long-term high-dose use, rare cardiac valve concerns.. Caber is way more potent and for most users is p5p dosing around 100-200mg enough on cycle. I would only recommend if bloodwork show still very high levels of prolactin even after p5p use.
Acne
Acne is a common side effect of anabolic-androgenic steroids because AAS increase androgen activity in the skin, which stimulates sebaceous glands to produce more oil. Higher sebum production, combined with changes in skin cell turnover and bacterial growth, can clog pores and lead to inflammatory acne. Ofc this also is really depending on how you respond.
Isotretinoin (Accutune)
Isotretinoin dramatically reduces the sebum production making it very effective for ‘roid acne’. Its unfortunate side effect is the significant stress on the liver it causes. Even if you already use compounds that have a negative impact on the liver like orals. Also it can have a positive influence on anti aging.
Liver protection
When people talk about liver stress from anabolic-androgenic steroids, they’re usually talking about oral compounds like methandrostenolone (Dianabol), oxandrolone (Anavar), stanozolol (Winstrol), oxymetholone (Anadrol) and Isotretinoin (Accutune) we mentioned before. The big issue is the 17-alpha-alkylated structure used in many oral AAS. With repeated exposure, this can elevate liver enzymes such as ALT and AST, indicating hepatocellular stress.
NAC
Nac also know as N-acetylcysteine is a commonly used as a supportive compound for the liver. NAC is a precursor to glutathione, one of the body’s main intracellular antioxidants. Glutathione helps neutralize reactive oxygen species and supports detoxification pathways in the liver helping it elp reduce oxidative stress and support liver enzyme. !!Note that this a supportive drug and not protective, that means it cant save your liver from 4 different stupidly doses orals.!!
Commun doses are around 600-1800mg depending on compounds and doses.
LDL/HDL issues
Some AAS may severely worsen cholesterol markes, by increasing LDL and suppressing HDL. LDL is also know as the ‘bad’ cholesterol and HDL as the ‘good’ one that clears LDL out. AAS use tends to tilt this scale aggressively in the wrong direction. Not what we want here comes:
Ezetimibe:
Ezetimibe reduces intestinal cholesterol absorption, which can help help with limiting LDL elevation. LDL Reduction: Typically reduces LDL by 15–22% when used alone. Its has only a very mild effect on HDL only increasing it by a few %.
Common used dosages are 10mg ed
Hairloss
The use of AAS turn you into to a bald chud gymcel, we for sure dont want this to happen. But how does it work? It really is depending on the compound. Hairloss mainly accors from DHT in males. DHT is very potent and has a high affinity for receptors in the scalp.
Once DHT binds to these receptors, it triggers a process called follicle miniaturization. The growth phase of the hair becomes shorter, the hair shaft becomes thinner, and eventually, the follicle stops producing hair entirely and falls out. Hairloss is mainly genetic and ofc it really matters if your hair is sensitive or not. Roiding can heavily speed up this process.
Finasteride and Dutasteride
There job is to inhibit the 5-alpha reductase (5-AR) enzyme, which is responsible for converting Testosterone into DHT. Inhibiting this could result in very low to non DHT what has some potential side effects like lower libido and mood. This is a very effective solution but is only interesting if you only take test. DHT derivatives are already structurally similar to DHT, the 5-AR enzyme has nothing to do with them. And 19-nor derivatives have zero influence on the 5-AE enzyme and taking Nandrolone can make it worse. Also wait until you are finished with puberty with taking this since DHT plays a big role in development.
RU58841
The magical solution if your not only taking test?
How does it work? Ru58841 is a topical androgen receptor antagonist used to local block the androgen activity at the hair follicle, helping with local blocking except eliminating DHT systemly and is very good if you take DHT derivatives or things like 19-nor derivatives.
(Minoxidil)
Bit water but wanted to still add this. Like most of you know minoxidil can be a very good hair growth supporting compound. It can be taken oral commonly used at 2.5mg. It works systemly can promote overall hair growth in like places like eyebrows or lashes. Only downside, it may result in bloating. Also topical available at like 5% at the scalp or other places (
Mirin pubes growth?)
Blood Pressure
maybe the most common side effect in the roiding world, elevated blood pressure. This happens because AAS stimulate EPO production, making the blood more viscous. Pumping thicker blood through the same size vessels requires significantly more force. Also Certain compounds keep the body in a fight or flight state, keeping the heart rate and blood pressure elevated even at rest. High blood pressure can cause many serious problems like: kidney damage, heart growth and increased risk for a stroke. To minimize cardiovascular strain you can do the following things:
Blood pressure monitoring
firstly its necessary to monitor yout blood pressure for any unwanted high blood pressure. I recommend monitoring daily at the start of your cycle and artillerys or at an increase in doses. If stable you can do it like 1-2x a week.
It doesn't really matter which one you get just order one of amazon for like 30 bucks.
Keep resting BP <120/80
Telmisartan
Telmisartan is a medication in the ARB (Angiotensin II Receptor Blocker) class. Besides doing a very good job at lowering bp by the angiotensin II receptor, but what makes it special is it other additional benefits such s vascular protection and improved insulin sensitivity what could be very good for people who use growth hormone/insulin. Commonly used doses are 40mg ed, but it is bp depended.
Tadalafil (Cialis)
While being primarily known for treating erectile dysfunction and improving sex performance, low-dose daily Tadalafil (2.5mg to 5mg) is commonly used for its cardiovascular benefits. Tadalafil is a PDE5 inhibitor. It works by increasing nitric oxide availability, which helps the inner lining of the blood vessels (the endothelium) function better. Using this two together can have an effective “stacking” effect on blood pressure.
Bloating
AAS have multiple ways to having lots of water bloating on cycle. Mainly through excessive E2 but we already talked about that. We havent about Aldosterone. Aldosterone is a steroid hormone produced by the adrenal glands that serves as the body’s primary regulator of salt and water balance. 19 nor’s have a special mechanism bind directly to mineralocorticoid receptors (MR) in the kidneys and they can act as Aldosterone.
Telmisartan
Yes again, this compound also has a very good influence on bloating too. Telmisartan blocks the Angiotensin II receptor. Since Angiotensin II is the signal that tells the body to release more aldosterone telmisartan can help decrease bloating. Also supporting the kidneys from AAS. This compound should be a must take on cycle.
Potassium
Having a good potassium-sodium ratio is very helpful.Aldosterone causes the body to dump potassium. By increasing your potassium intake through food you help counteract the sodium-retaining effects of aldosterone and encourage the kidneys to flush excess fluid.
Diuretics
Using diuretics can be a very good tool for minimizing bloat on cycle. Its a very effective method getting rid of bloat but it can have some side effects. The biggest risk of using any diuretic on cycle is that AAS already increase your red blood cell count. If you use a diuretic to flush out water, your blood becomes even thicker (concentrated). This significantly increases the risk of blood clots and strokes, especially if you are not monitoring your levels.
Eplerenone
Eplerenone works by blocking aldosterone from binding to its receptors in the kidneys. As we discussed, aldosterone is the hormone responsible for making you hold onto salt and water while flushing out potassium. By blocking this signal, Eplerenone helps your body dump excess water and sodium, which directly lowers blood pressure and reduces bloating. Overall a great compound common dosed around 25mg ed.
Indapamide
Indapamide is a thiazide-like diuretic. What makes Indapamide unique is its dual mechanism of action it acts as both a mild diuretic and a direct vasodilator (relaxing blood vessel walls). While being a milder diuretic it is also great in lowering bp. Doses are between 1.5-2.5mg ed
Melatonin
While it is mainly being used at doses between 0.5mg - 5mg for sleep and regulating circadian rhythm high dosages is often used for its potent antioxidant and anti-inflammatory properties. High doses are sometimes researched for their ability to protect cells from oxidative stress, particularly in the brain and heart. This can be extremely helpful in context of liver/neuroprotecting benefits. Dosages may 20mg to 300mg+. While this is still experimental it seems promising.
PCT
When on cycle the body suppresses natural hormone release. The hypothalamus reduces gonadotropin-releasing hormone (GnRH), the pituitary lowers luteinizing hormone (LH) and follicle-stimulating hormone (FSH), and the testes reduce endogenous testosterone production. After cycle you want your balls to work again so you can spread your seed to put a other low iq subhuman on the earth just like you. So how do we do this? Our goal is is to accelerate recovery of LH and FSH secretion so that testosterone production resumes more quickly.
HCG
hCG is a glycoprotein hormone structurally very similar to luteinizing hormone (LH). It binds directly to LH receptors in the testes. Main uses are prevent shrinking of your balls during cycle or before SERM-based therapy to restore testicular responsiveness in time where your esters are running out.
During cycle common doses are: 250IU 3x a week or 250IU eod
Tamoxifen
Tamoxifen is a Selective Estrogen Receptor Modulator (SERM). It acts as an estrogen receptor antagonist in the hypothalamus and pituitary. By blocking estrogen’s negative feedback, it increases GnRH release from the hypothalamus. This stimulates the pituitary to produce more LH and FSH.
PCT doses: 10-40mg daily
Clomiphene
Like tamoxifen, clomiphene blocks estrogen receptors in the hypothalamus and pituitary. This reduces estrogen-mediated negative feedback and increases GnRH secretion, which raises LH and FSH levels. It is way stronger in increasing GnRh secretion then Tamoxifen
The start of PCT is very depending on the ester of your compound. Its very important that you start after 3-4 halflives of your ester. If exogenous androgens are still high enough to strongly suppress the axis, SERMs won’t be very effective because negative feedback is still active.
After one half-life = 50% remaining
After two half-lives = 25% remaining
After three half-lives = 12.5% remaining
After four half-lives = 6.25% remaining
Bloodwork
a very important thing if you want to safely run a cycle. There are 3 types
1. Pre cycle: This is before you start your cycle. This gives the baseline and is very helpful further on.
2. Mid cycle, mosly done 4-6 weeks on cycle. Here you check if your gear is legit, E2 levels and adjust any other doses based on health markers.
3. Pre PCT Bloodwork, mostly done 4-6 week after PCT is finished. Here you check your LH, FSH, E2 and test levels too see if they are back in check from before cycle.
!!Testosterone recovery does not guarantee sperm recovery. If fertility matters, semen analysis is required.!!
Now that we have gone over the basics over side management in AAS uses here is mine for those interested:
Mirin pubes growth?)Blood Pressure
maybe the most common side effect in the roiding world, elevated blood pressure. This happens because AAS stimulate EPO production, making the blood more viscous. Pumping thicker blood through the same size vessels requires significantly more force. Also Certain compounds keep the body in a fight or flight state, keeping the heart rate and blood pressure elevated even at rest. High blood pressure can cause many serious problems like: kidney damage, heart growth and increased risk for a stroke. To minimize cardiovascular strain you can do the following things:
Blood pressure monitoring
firstly its necessary to monitor yout blood pressure for any unwanted high blood pressure. I recommend monitoring daily at the start of your cycle and artillerys or at an increase in doses. If stable you can do it like 1-2x a week.
It doesn't really matter which one you get just order one of amazon for like 30 bucks.
Keep resting BP <120/80
Telmisartan
Telmisartan is a medication in the ARB (Angiotensin II Receptor Blocker) class. Besides doing a very good job at lowering bp by the angiotensin II receptor, but what makes it special is it other additional benefits such s vascular protection and improved insulin sensitivity what could be very good for people who use growth hormone/insulin. Commonly used doses are 40mg ed, but it is bp depended.
Tadalafil (Cialis)
While being primarily known for treating erectile dysfunction and improving sex performance, low-dose daily Tadalafil (2.5mg to 5mg) is commonly used for its cardiovascular benefits. Tadalafil is a PDE5 inhibitor. It works by increasing nitric oxide availability, which helps the inner lining of the blood vessels (the endothelium) function better. Using this two together can have an effective “stacking” effect on blood pressure.
Bloating
AAS have multiple ways to having lots of water bloating on cycle. Mainly through excessive E2 but we already talked about that. We havent about Aldosterone. Aldosterone is a steroid hormone produced by the adrenal glands that serves as the body’s primary regulator of salt and water balance. 19 nor’s have a special mechanism bind directly to mineralocorticoid receptors (MR) in the kidneys and they can act as Aldosterone.
Telmisartan
Yes again, this compound also has a very good influence on bloating too. Telmisartan blocks the Angiotensin II receptor. Since Angiotensin II is the signal that tells the body to release more aldosterone telmisartan can help decrease bloating. Also supporting the kidneys from AAS. This compound should be a must take on cycle.
Potassium
Having a good potassium-sodium ratio is very helpful.Aldosterone causes the body to dump potassium. By increasing your potassium intake through food you help counteract the sodium-retaining effects of aldosterone and encourage the kidneys to flush excess fluid.
Diuretics
Using diuretics can be a very good tool for minimizing bloat on cycle. Its a very effective method getting rid of bloat but it can have some side effects. The biggest risk of using any diuretic on cycle is that AAS already increase your red blood cell count. If you use a diuretic to flush out water, your blood becomes even thicker (concentrated). This significantly increases the risk of blood clots and strokes, especially if you are not monitoring your levels.
Eplerenone
Eplerenone works by blocking aldosterone from binding to its receptors in the kidneys. As we discussed, aldosterone is the hormone responsible for making you hold onto salt and water while flushing out potassium. By blocking this signal, Eplerenone helps your body dump excess water and sodium, which directly lowers blood pressure and reduces bloating. Overall a great compound common dosed around 25mg ed.
Indapamide
Indapamide is a thiazide-like diuretic. What makes Indapamide unique is its dual mechanism of action it acts as both a mild diuretic and a direct vasodilator (relaxing blood vessel walls). While being a milder diuretic it is also great in lowering bp. Doses are between 1.5-2.5mg ed
Melatonin
While it is mainly being used at doses between 0.5mg - 5mg for sleep and regulating circadian rhythm high dosages is often used for its potent antioxidant and anti-inflammatory properties. High doses are sometimes researched for their ability to protect cells from oxidative stress, particularly in the brain and heart. This can be extremely helpful in context of liver/neuroprotecting benefits. Dosages may 20mg to 300mg+. While this is still experimental it seems promising.
PCT
When on cycle the body suppresses natural hormone release. The hypothalamus reduces gonadotropin-releasing hormone (GnRH), the pituitary lowers luteinizing hormone (LH) and follicle-stimulating hormone (FSH), and the testes reduce endogenous testosterone production. After cycle you want your balls to work again so you can spread your seed to put a other low iq subhuman on the earth just like you. So how do we do this? Our goal is is to accelerate recovery of LH and FSH secretion so that testosterone production resumes more quickly.
HCG
hCG is a glycoprotein hormone structurally very similar to luteinizing hormone (LH). It binds directly to LH receptors in the testes. Main uses are prevent shrinking of your balls during cycle or before SERM-based therapy to restore testicular responsiveness in time where your esters are running out.
During cycle common doses are: 250IU 3x a week or 250IU eod
Tamoxifen
Tamoxifen is a Selective Estrogen Receptor Modulator (SERM). It acts as an estrogen receptor antagonist in the hypothalamus and pituitary. By blocking estrogen’s negative feedback, it increases GnRH release from the hypothalamus. This stimulates the pituitary to produce more LH and FSH.
PCT doses: 10-40mg daily
Clomiphene
Like tamoxifen, clomiphene blocks estrogen receptors in the hypothalamus and pituitary. This reduces estrogen-mediated negative feedback and increases GnRH secretion, which raises LH and FSH levels. It is way stronger in increasing GnRh secretion then Tamoxifen
The start of PCT is very depending on the ester of your compound. Its very important that you start after 3-4 halflives of your ester. If exogenous androgens are still high enough to strongly suppress the axis, SERMs won’t be very effective because negative feedback is still active.
After one half-life = 50% remaining
After two half-lives = 25% remaining
After three half-lives = 12.5% remaining
After four half-lives = 6.25% remaining
Bloodwork
a very important thing if you want to safely run a cycle. There are 3 types
1. Pre cycle: This is before you start your cycle. This gives the baseline and is very helpful further on.
2. Mid cycle, mosly done 4-6 weeks on cycle. Here you check if your gear is legit, E2 levels and adjust any other doses based on health markers.
3. Pre PCT Bloodwork, mostly done 4-6 week after PCT is finished. Here you check your LH, FSH, E2 and test levels too see if they are back in check from before cycle.
!!Testosterone recovery does not guarantee sperm recovery. If fertility matters, semen analysis is required.!!
Now that we have gone over the basics over side management in AAS uses here is mine for those interested:
Orals | dosage | reason | time |
T3 | 20mcg ed | tren damages t4 -> t3 | morning |
Telmisartan | 40mg ed -> bp dep. | Blood pressure, vascular protection | morning |
P5P | 100mg ed | prolactin | morning |
Isotretin | 10mg ed -> dep. | acne | with large meal |
Tadanafil | 5 mg ed | bp, blood flow | morning |
Nac | 1200mg ed | Liver Support | not with heavy meals |
Melatonin | 5mg ed | Sleep | before sleep |
Ezetimibe | 10mg ed | Lowers LDL cholesterol | evening |
Indaparamide | 1.5mg ed | bloat + bp lowering | morning |
Ofc i want to mention that beside taking a few orals overall healthy habits are just as important. Limit alcohol and smoking, eat fat fish or 2g EPA/DHA, optimal sleep and eat well.
Thank you for reading this guide at the end i want to leave a disclaimer that these are medications and will not fully counter sides or making AAS use safe. If you are taking roids you must acknowledge the facts that there are side effects and there are risks and taking a few orals wont magically solve all the damage to your body. I Surely dont recommend using roids if you don't know what you are doing
Thanks for reading lmk if you have any tips to do better next time.
(and yes i did write this all myself rep pls)
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