Guide Why SARMs are dogshit (8 Viewers)

[Biomaxx]

He also thinks high dose dht derivatives will grow your bones and that 5 ar blockers will make u lose bone mass, fyi he can’t even comprehend the difference between subjective and objective and says the side effects on roids are “subjective” as he js uses the word as buzzword cause he doesn’t understand what interindividual variability is

Subjective roid sides jfl

 

[Biomaxx]

Kindaaaa not feeling hair loss on this cycle

 

[Daker]

Kindaaaa not feeling hair loss on this cycle

tren making niggas gay is objective verifiable info btw.

 

[Biomaxx]

tren making niggas gay is objective verifiable info btw.

Tren made me super gay

 

[dysregulated]

Tren made me super gay

hi lol

 

[Daker]

Tren made me super gay

Made Mandy super gay too for what i know

 

[Biomaxx]

Made Mandy super gay too for what i know

Mandy was gay before tren lol
I asked

 

[Biomaxx]

hi lol

Dms

 

[dysregulated]

Mandy was gay before tren lol
I asked

didnt know mandy was gay

 

[Daker]

didnt know mandy was gay

Wasn't it pretty fucking obvious? he flirted innapropiately with every men here and would be very open about it.

 

[dysregulated]

Wasn't it pretty fucking obvious? he flirted innapropiately with every men here and would be very open about it.

well i didnt have internet when he was active, i only knew him for the last 2 weeks of his ldar

 

[birthdefect]

@birthdefect why were you agreeing with his enclo as a test base thing nigga? you are supposed to be smart

because i know very little about the more niche stuff of the negative feedback loop
in my mind it doesnt even matter whether its a sarm or steroid at a low dose
my thinking was that the estrogen and androgen receptors in the brain act as a way to signal the total amount of sex steroids, so if you antagonise estrogen receptors you have a greater window to agonise the androgen receptors, so a low dose sarm or steroid like oxandrolone or halotestin would fit in that window

thats just my thinking tho, this thread is mostly on the non tissue selectivity of sarms
the real sarm we need would function just like enclo but for androgen receptors instead of estrogen receptors, then everything would be great

 

[Daker]

because i know very little about the more niche stuff of the negative feedback loop
in my mind it doesnt even matter whether its a sarm or steroid at a low dose
my thinking was that the estrogen and androgen receptors in the brain act as a way to signal the total amount of sex steroids, so if you antagonise estrogen receptors you have a greater window to agonise the androgen receptors, so a low dose sarm or steroid like oxandrolone or halotestin would fit in that window

thats just my thinking tho, this thread is mostly on the non tissue selectivity of sarms
the real sarm we need would function just like enclo but for androgen receptors instead of estrogen receptors, then everything would be great

It doesn't work like that, read the thread. "uhmm" what? SARMs are dogshit.

 

[birthdefect]

It doesn't work like that, read the thread. "uhmm" what? SARMs are dogshit.

the premise here is that the androgen receptors are fully saturated
i was thinking a very low dose to avoid that saturation specifically
could that still not be done?

 

[Daker]

View attachment 54352
the premise here is that the androgen receptors are fully saturated
i was thinking a very low dose to avoid that saturation specifically
could that still not be done?

No,even a micro dose fails because the HPTA doesn't require a full 100% receptor saturation to shut down or to be downregulated, AAS and SARMs will aggressively bind to the ARs in the hypothalamus and downregulate the pulsatile release of GnRH.