[Biomaxx]

Pwease

 

[Mandy]

Never experienced any water retention on roids ever since im not prone to any water retention,or it could just be the mild diuretic effect of the copious amounts of caffeine I consume. But generally bloat from GH is the effect of the higher insulin release of the pancreas which causes sodium reuptake via kidneys. So generally very water advice but reducing sodium is indeed effective in that matter.

 

[certified_LTN]

i would guess Atrial Natriuretic Peptides, but im stupid in this things

 

[Godveil Heir]

Never experienced any water retention on roids ever since im not prone to any water retention,or it could just be the mild diuretic effect of the copious amounts of caffeine I consume. But generally bloat from GH is the effect of the higher insulin release of the pancreas which causes sodium reuptake via kidneys. So generally very water advice but reducing sodium is indeed effective in that matter.

gh primarily drives sodium and water retention through direct renal effects like ENaC activation, Na+/K+-ATPase upregulation, renin elevation, ANF suppression rather than mainly via pancreatic insulin spikes.
Biomaxx I think you can use Nebivolol
it suppresses renin release (beta-1 blockade drops PRA ~50%) + beta-3 agonism activates endothelial eNOS, boosting NO for vasodilation and improved renal perfusion

 

[Biomaxx]

Needed to look into this but forgot. Holy shit this took me a while to acc understand. Ill look through my sources see what I can find. Ty

gh primarily drives sodium and water retention through direct renal effects like ENaC activation, Na+/K+-ATPase upregulation, renin elevation, ANF suppression rather than mainly via pancreatic insulin spikes.
Biomaxx I think you can use Nebivolol
it suppresses renin release (beta-1 blockade drops PRA ~50%) + beta-3 agonism activates endothelial eNOS, boosting NO for vasodilation and improved renal perfusion

 

[Mandy]

gh primarily drives sodium and water retention through direct renal effects like ENaC activation, Na+/K+-ATPase upregulation, renin elevation, ANF suppression rather than mainly via pancreatic insulin spikes.
Biomaxx I think you can use Nebivolol
it suppresses renin release (beta-1 blockade drops PRA ~50%) + beta-3 agonism activates endothelial eNOS, boosting NO for vasodilation and improved renal perfusion

My bad. But dosent generally the pancreatic insulin spikes also play a role even if it’s partially? I gotta get more research into the kidneys,it’s one of the organs I pretty much don’t know much about compared to everything else.