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First of all, the ONLY way to grow past your genetic boundaries is too prolong your height growth window. The way to do this is to delay you epiphyseal plates from ossificating (the procces of cartilage turning into bone) the way this happens is when chondrocytes ( the main cell in cartilage) proliferate, they then hypertrophy causing the calcification of the area. This lays down a foundation for the drivers of that ossifcation (osteoclast and osteoblast) to start working.
But daddy ? How can i stop this?
dont worry my child, these stacks ive formulated will include compunds that do just that and ill explain how
I could probably come up with more but I cba and I have work in an hour.
Gonna do some more research into calcium blockades but I didn't include them bc they seem stupid asf
Much love bhais please correct me of im wrong and leave your own suggestions
Obv these can be swapped around js listing the conpounds really
9Dino
Mandy
Ogre_cel
But daddy ? How can i stop this?
dont worry my child, these stacks ive formulated will include compunds that do just that and ill explain how
This first stack is comprised of four compund
Hgh
aromasin
abaloparatide
t4
hgh-
everyone knows abt hgh so i wont do much explaing. Exougenous hgh follows the bodies natural process (also why it mogs gh secretagogues and increlex). The main takeaway is that it follows the process of igf signaling also producing adequate igfpb and other important allies.
aromasin-
aromasin is the absolute best aromatase inhibitor. Its not harsh so it doest immediatly crash levels. CRUCIAL- never take high dose ai. It may seem like a good idea, less e2= less fusure . But at the cost of mental retardation and stunted deveolpment and a high potential of plate fractures. Micro dosing 6.25mg eod should be taken
abaloparatide (DYOR
)-
Ive included abaloparatide here for two reason.
It grows bone- as its a pthrp its main goal is to grow bone (prescribed for osteoporosis) meaning a couple extra cm could be expected
It can delay plate fusure (unproven) - theoretically it can
Slow proliferating chondrocyte maturation
Slow the chondrocytes reaching that hypertrophic state (only if taken early in puberty)
t4-
Thyroid hormone primes tissues to respond to GH. It increases the expression of GH receptors and boosts the liver's ability to produce IGF-1-the real growth driver downstream of GH. If thyroid hormone is low, GH may be released normally, but the body is basically deaf to it. When T4 is too high, GH and IGF-1 can be high, but the system burns so hot that the net effect becomes chaotic: growth plate exhaustion, acceleration of bone turnover, and metabolic frenzy.
Hgh
aromasin
abaloparatide
t4
hgh-
everyone knows abt hgh so i wont do much explaing. Exougenous hgh follows the bodies natural process (also why it mogs gh secretagogues and increlex). The main takeaway is that it follows the process of igf signaling also producing adequate igfpb and other important allies.
aromasin-
aromasin is the absolute best aromatase inhibitor. Its not harsh so it doest immediatly crash levels. CRUCIAL- never take high dose ai. It may seem like a good idea, less e2= less fusure . But at the cost of mental retardation and stunted deveolpment and a high potential of plate fractures. Micro dosing 6.25mg eod should be taken
abaloparatide (DYOR
)-Ive included abaloparatide here for two reason.
It grows bone- as its a pthrp its main goal is to grow bone (prescribed for osteoporosis) meaning a couple extra cm could be expected
It can delay plate fusure (unproven) - theoretically it can
Slow proliferating chondrocyte maturation
Slow the chondrocytes reaching that hypertrophic state (only if taken early in puberty)
t4-
Thyroid hormone primes tissues to respond to GH. It increases the expression of GH receptors and boosts the liver's ability to produce IGF-1-the real growth driver downstream of GH. If thyroid hormone is low, GH may be released normally, but the body is basically deaf to it. When T4 is too high, GH and IGF-1 can be high, but the system burns so hot that the net effect becomes chaotic: growth plate exhaustion, acceleration of bone turnover, and metabolic frenzy.
Hgh
aromasin
insulin
CNP analogs
Hgh and aromsin explained
insulin-
It’s one of the strongest IGF-1 supporters because the liver needs adequate insulin signaling to make IGF-1 in response to GH. Low insulin states (fasting, uncontrolled diabetes) mean GH goes up, but IGF-1 goes down. lots of signal but no growth. High insulin states (post-meal, insulin therapy) make IGF-1 output more robust, which is one reason puberty, when insulin is often tr higher, becomes a prime time for growth spurts
Cnp analogs-
CNP maintains the functionality of the growth plate by keeping chondrocytes in their proliferative state and acting against FGFR3 signalling, which normally limits the growth of the bone. The CNP analogues, like vosoritide, extend this effect by having a longer half-life. In achondroplasia, such analogues increase the velocity of growth by strengthening the proliferative signals in the growth plate. They do not directly delay the normal epiphyseal closure but are theoretically discussed since they are capable of prolonging the proliferative phase of chondrocytes.
aromasin
insulin
CNP analogs
Hgh and aromsin explained
insulin-
It’s one of the strongest IGF-1 supporters because the liver needs adequate insulin signaling to make IGF-1 in response to GH. Low insulin states (fasting, uncontrolled diabetes) mean GH goes up, but IGF-1 goes down. lots of signal but no growth. High insulin states (post-meal, insulin therapy) make IGF-1 output more robust, which is one reason puberty, when insulin is often tr higher, becomes a prime time for growth spurts
Cnp analogs-
CNP maintains the functionality of the growth plate by keeping chondrocytes in their proliferative state and acting against FGFR3 signalling, which normally limits the growth of the bone. The CNP analogues, like vosoritide, extend this effect by having a longer half-life. In achondroplasia, such analogues increase the velocity of growth by strengthening the proliferative signals in the growth plate. They do not directly delay the normal epiphyseal closure but are theoretically discussed since they are capable of prolonging the proliferative phase of chondrocytes.
I could probably come up with more but I cba and I have work in an hour.
Gonna do some more research into calcium blockades but I didn't include them bc they seem stupid asf
Much love bhais please correct me of im wrong and leave your own suggestions
Obv these can be swapped around js listing the conpounds really
9Dino
Mandy
Ogre_cel
