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Looksmax I'm unsure

Hunterr

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I've read about Gh, test , igf-1, dht blockers, estrogens blockers, osteoblast/osteoclast, peptides, sclerosis inhibition and many others. But wich is the best stack to do for height growth and facial development??
Please answer
 

Dexter

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dht contributes to facial dimorphism and he said facial development and height growth only not overall aesthetics

also hes prolly in puberty if hes asking ts
DHT neither masculinizes your face nor does it contribute to height growth

the only manliness you'll see will be a shining bald head
 
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DHT neither masculinizes your face nor does it contribute to height growth

the only manliness you'll see will be a shining bald head
bhai anything with a high androgenic rating will masculinise cuz it binds to all androgen receptors provided hes young enough (regarding actual bone changes)

dht is actually quite essential in development compared to later ages
 

Dexter

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bhai anything with a high androgenic rating will masculinise cuz it binds to all androgen receptors provided hes young enough (regarding actual bone changes)

dht is actually quite essential in development compared to later ages
IMG_20251202_224511.jpg

Conclusion: Patients with dihydrotestosterone deficiency present normal bone mineral density, suggesting that dihydrotestosterone is not the main androgen acting in bone.


4843552_IMG_3145.jpeg


The two on the left quite literally have 0 DHT in their body yet they are of similar height/skeletal characteristics as the other person.
It's testosterone, E2, hGH, IGF1 that promote bone growth and not DHT.

chondrocyte proliferation + hypertrophy in the growth plates are run by the GH -> hepatic and local IGF1 axis. IGF1 is the proximate mitogen that stimulates chondrocyte proliferation, matrix production and hypertrophic differentiation which ACTUALLY matters in terms of height growtg. GH also increases local IGF1 production in the growth plates.

test acting via AR will only stimulate periosteal apposition during puberty producing larger bone diameter and different bone geometry in guys
the thing is some of testosterone’s bone effects are mediated indirectly after aromatization to E2 (which also affects cortical or trabecular compartments and bone turnover. and guess what? dht cannot be aromatized.
 

Cheat

Pain is Beauty #cheattowin
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View attachment 10710
Conclusion: Patients with dihydrotestosterone deficiency present normal bone mineral density, suggesting that dihydrotestosterone is not the main androgen acting in bone.


View attachment 10711


The two on the left quite literally have 0 DHT in their body yet they are of similar height/skeletal characteristics as the other person.
It's testosterone, E2, hGH, IGF1 that promote bone growth and not DHT.

chondrocyte proliferation + hypertrophy in the growth plates are run by the GH -> hepatic and local IGF1 axis. IGF1 is the proximate mitogen that stimulates chondrocyte proliferation, matrix production and hypertrophic differentiation which ACTUALLY matters in terms of height growtg. GH also increases local IGF1 production in the growth plates.

test acting via AR will only stimulate periosteal apposition during puberty producing larger bone diameter and different bone geometry in guys
the thing is some of testosterone’s bone effects are mediated indirectly after aromatization to E2 (which also affects cortical or trabecular compartments and bone turnover. and guess what? dht cannot be aromatized.
too much effort for a reply mate
 
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View attachment 10710
Conclusion: Patients with dihydrotestosterone deficiency present normal bone mineral density, suggesting that dihydrotestosterone is not the main androgen acting in bone.


View attachment 10711


The two on the left quite literally have 0 DHT in their body yet they are of similar height/skeletal characteristics as the other person.
It's testosterone, E2, hGH, IGF1 that promote bone growth and not DHT.

chondrocyte proliferation + hypertrophy in the growth plates are run by the GH -> hepatic and local IGF1 axis. IGF1 is the proximate mitogen that stimulates chondrocyte proliferation, matrix production and hypertrophic differentiation which ACTUALLY matters in terms of height growtg. GH also increases local IGF1 production in the growth plates.

test acting via AR will only stimulate periosteal apposition during puberty producing larger bone diameter and different bone geometry in guys
the thing is some of testosterone’s bone effects are mediated indirectly after aromatization to E2 (which also affects cortical or trabecular compartments and bone turnover. and guess what? dht cannot be aromatized.
mirin iqmogged me
 
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