why would he not block DHT unless he's in pubertyhgh+ai and dont block dht
dht contributes to facial dimorphism and he said facial development and height growth only not overall aestheticswhy would he not block DHT unless he's in puberty
DHT neither masculinizes your face nor does it contribute to height growthdht contributes to facial dimorphism and he said facial development and height growth only not overall aesthetics
also hes prolly in puberty if hes asking ts
bhai anything with a high androgenic rating will masculinise cuz it binds to all androgen receptors provided hes young enough (regarding actual bone changes)DHT neither masculinizes your face nor does it contribute to height growth
the only manliness you'll see will be a shining bald head
bhai anything with a high androgenic rating will masculinise cuz it binds to all androgen receptors provided hes young enough (regarding actual bone changes)
dht is actually quite essential in development compared to later ages
too much effort for a reply mateView attachment 10710
Conclusion: Patients with dihydrotestosterone deficiency present normal bone mineral density, suggesting that dihydrotestosterone is not the main androgen acting in bone.
View attachment 10711
The two on the left quite literally have 0 DHT in their body yet they are of similar height/skeletal characteristics as the other person.
It's testosterone, E2, hGH, IGF1 that promote bone growth and not DHT.
chondrocyte proliferation + hypertrophy in the growth plates are run by the GH -> hepatic and local IGF1 axis. IGF1 is the proximate mitogen that stimulates chondrocyte proliferation, matrix production and hypertrophic differentiation which ACTUALLY matters in terms of height growtg. GH also increases local IGF1 production in the growth plates.
test acting via AR will only stimulate periosteal apposition during puberty producing larger bone diameter and different bone geometry in guys
the thing is some of testosterone’s bone effects are mediated indirectly after aromatization to E2 (which also affects cortical or trabecular compartments and bone turnover. and guess what? dht cannot be aromatized.
can't really convince someone just by saying "no, dht does not cause bone growth"too much effort for a reply mate
knowledge mogged him though, mirincan't really convince someone just by saying "no, dht does not cause bone growth"
mirin iqmogged meView attachment 10710
Conclusion: Patients with dihydrotestosterone deficiency present normal bone mineral density, suggesting that dihydrotestosterone is not the main androgen acting in bone.
View attachment 10711
The two on the left quite literally have 0 DHT in their body yet they are of similar height/skeletal characteristics as the other person.
It's testosterone, E2, hGH, IGF1 that promote bone growth and not DHT.
chondrocyte proliferation + hypertrophy in the growth plates are run by the GH -> hepatic and local IGF1 axis. IGF1 is the proximate mitogen that stimulates chondrocyte proliferation, matrix production and hypertrophic differentiation which ACTUALLY matters in terms of height growtg. GH also increases local IGF1 production in the growth plates.
test acting via AR will only stimulate periosteal apposition during puberty producing larger bone diameter and different bone geometry in guys
the thing is some of testosterone’s bone effects are mediated indirectly after aromatization to E2 (which also affects cortical or trabecular compartments and bone turnover. and guess what? dht cannot be aromatized.


