Guide HOW TO START YOUR FIRST CYCLE 101 (for bodybuilding) (17 Viewers)

[looksmin]

FIRST CYCLE 101
(for bodybuilding)

Most guys jump in WAY too fast and end up with more problems than gains.
A proper first cycle is simple, one compound (test ofc), moderate dosage (200-300mg), proper support, and bloodwork (nothing ever happens jfl)

The goal here isn't to get huge in 8 fucking weeks if you think this is possible just log off and rope; It's to learn how your body RESPONDS while keeping this as safe as possible.

COMPOUNDS YOU SHOULD USE
TEST ONLY YOU RETARD. Some people hate on it but in reality it's the safest way of doing things.
Test E or C is easy and good, it's what your body already knows how to use, the sides are predictable, and the gains are real.
Stay away from orals, tren, deca, or any fancy stacks you see your favorite experienced IFBB Pro using on your first go.

DOSING, LENGTH & SUPPORT
200-400MG of Test per week is enough. Run for 12-16 weeks so you actually see what it can do and how YOU RESPOND. I personally prefer pinning everyday to mitigate fluctuations as much as possible. If your not pinning ED what are you doing jfl.
AI (aromatase inhibitor) example I use - Arimidex. if you're prone to estrogen sides, get bloods to know for sure.
A recommended dose of arimidex is 0.5mg a day, that is what I currently use to keep my e2 low and not fully crashed. If you crash your e2 levels completely during puberty you're in for a fucking treat. Make sure you're taking supplements to keep your liver in check. Examples of this include; NAC or TUDCA, and Milk thistle. Take your daily vitamins as well. These include; Vitamin D3, Vitamin K2, Magnesium Glycinate, Boron, Zinc, etc.

WHAT TO EXPECT
On a solid first cycle with dialed in training, diet + sleep, most guys put on 10-15 lbs total (maybe even less it all depends on how you respond), with roughly half of that being keep able.
You'll look fuller, recover quicker, and feel better inside the gym but you will also get some water retention and possible acne if not mitigated properly.

SUMMARY
A first cycle is not about going as hard as possible, it's about learning your body with the safest, most effective compound (testosterone).
Keep the dose moderate (200-400 mg/week) run it long enough to see real results and get your bloods done!

No this picture above is not me. It's Some random guy on tiktok jfl​

 

[Dragon]

FIRST CYCLE 101
(for bodybuilding)

Most guys jump in WAY too fast and end up with more problems than gains.
A proper first cycle is simple, one compound (test ofc), moderate dosage (200-300mg), proper support, and bloodwork (nothing ever happens jfl)

The goal here isn't to get huge in 8 fucking weeks if you think this is possible just log off and rope; It's to learn how your body RESPONDS while keeping this as safe as possible.
View attachment 50303

COMPOUNDS YOU SHOULD USE
TEST ONLY YOU RETARD. Some people hate on it but in reality it's the safest way of doing things.
Test E or C is easy and good, it's what your body already knows how to use, the sides are predictable, and the gains are real.
Stay away from orals, tren, deca, or any fancy stacks you see your favorite experienced IFBB Pro using on your first go.
View attachment 50309

DOSING, LENGTH & SUPPORT
200-400MG of Test per week is enough. Run for 12-16 weeks so you actually see what it can do and how YOU RESPOND. I personally prefer pinning everyday to mitigate fluctuations as much as possible. If your not pinning ED what are you doing jfl.
AI (aromatase inhibitor) example I use - Arimidex. if you're prone to estrogen sides, get bloods to know for sure.
A recommended dose of arimidex is 0.5mg a day, that is what I currently use to keep my e2 low and not fully crashed. If you crash your e2 levels completely during puberty you're in for a fucking treat. Make sure you're taking supplements to keep your liver in check. Examples of this include; NAC or TUDCA, and Milk thistle. Take your daily vitamins as well. These include; Vitamin D3, Vitamin K2, Magnesium Glycinate, Boron, Zinc, etc.
View attachment 50310

WHAT TO EXPECT
On a solid first cycle with dialed in training, diet + sleep, most guys put on 10-15 lbs total (maybe even less it all depends on how you respond), with roughly half of that being keep able.
You'll look fuller, recover quicker, and feel better inside the gym but you will also get some water retention and possible acne if not mitigated properly.
View attachment 50312
SUMMARY
A first cycle is not about going as hard as possible, it's about learning your body with the safest, most effective compound (testosterone).
Keep the dose moderate (200-400 mg/week) run it long enough to see real results and get your bloods done!
View attachment 50308
No this picture above is not me. It's Some random guy on tiktok jfl​

no im doing extreme low dose test and tren first cycle

 

[looksmin]

last guide for today, this is mostly water if you have been on forums for long enough and know what you're doing with compounds and AAS'. Although just wanted to have this out here for any people who are going to make the mistake on running multiple compounds for their first cycle.

I also want to state that HGH paired with test is a great combination for your first cycle even tho HGH isn't an AAS they both work perfect paired togther.

 

[User123]

Just get an AI to make a cycle for u and up the doses it gives u 2x

 

[Syna]

missed dutasteride or KX-826 as a topical 5ar blocker and isotretinoin, topical E3 and tazarotene should be used anyways.

 

[looksmin]

missed dutasteride or KX-826 as a topical 5ar blocker and isotretinoin, topical E3 and tazarotene should be used anyways.

yea, didn't really go in depth on hair care or acne prevention. thought it would be common sense but not everyone needs hair protection on a test only cycle unless they're already norwood 2+ or have signs of balding imo


also fin > dut

 

[Syna]

yea, didn't really go in depth on hair care or acne prevention. thought it would be common sense but not everyone needs hair protection on a test only cycle unless they're already norwood 2+ or have signs of balding imo


also fin > dut

The point of 5ar blockers is prevention, no point using 5ar blockers when you already got raped by dht, why finasteride over dutasteride jfl?
If it's a guide you can't assume everyone already knows stuff.

 

[looksmin]

The point of 5ar blockers is prevention, no point using 5ar blockers when you are already raped by dht, why finasteride over dutasteride jfl?

well, for MOST guys 1mg of finasteride daily will be more then enough to significantly slow down and or stop hair loss from androgenic alopecia. i dont think anyone should start on dut and completely nuke their DHT on go, the argument can be made for some guys that have an aggressive form of androgenic alopecia that started early on but even for those guys 1mg of fin might be enough. Also you shouldn't use DUT at such an early age because in puberty, males who have no DHT in puberty lose masculization in their face and lose size in their penile region. anyone before 20 shouldn't hop on fin or dut imo, but if your hairline is fucked at 16 use ru-58841 or kx-826

 

[the wizard]

missed dutasteride or KX-826 as a topical 5ar blocker and isotretinoin, topical E3 and tazarotene should be used anyways.

also missed enclo for dimorphism

 

[Syna]

well, for MOST guys 1mg of finasteride daily will be more then enough to significantly slow down and or stop hair loss from androgenic alopecia. i dont think anyone should start on dut and completely nuke their DHT on go, the argument can be made for some guys that have an aggressive form of androgenic alopecia that started early on but even for those guys 1mg of fin might be enough. Also you shouldn't use DUT at such an early age because in puberty, males who have no DHT in puberty lose masculization in their face and lose size in their penile region. anyone before 20 shouldn't hop on fin or dut imo, but if your hairline is fucked at 16 use ru-58841 or kx-826

penile development stops at around age 16-18, DHT serves no other purpose but penile enlargement, after 18 you don't need it, there's no good argument for not using dutasteride, DHT rapes your hair follicles and your skin elasticity, specially when you are using supraphysiological amounts of androgens in this case test.

 

[ogre]

Ive been on 300mg test with no ai (trying to close my growth plates) for a month and gained 15lb

Just felt like sharing that info

 

[looksmin]

penile development stops at around age 16-18, DHT serves no other purpose but penile enlargement, after 18 you don't need it, there's no good argument for not using dutasteride, DHT rapes your hair follicles and your skin elasticity, specially when you are using supraphysiological amounts of androgens in this case test.

yes, but if you're in that age range of 15-18 and on AAS, i'd recommend either hopping on a topical hair prevention rather then going to DHT or FIN cause penile length / growth is law bro, can't have the foids with a 4 or 5 incher. i mean it all comes down to personal preference at the end of the day but if you're not experiencing significant hair loss / balding patterns and you're 15-18 i'd just stick to ru-58841 or KX-826

 

[Syna]

yes, but if you're in that age range of 15-18 and on AAS, i'd recommend either hopping on a topical hair prevention rather then going to DHT or FIN cause penile length / growth is law bro, can't have the foids with a 4 or 5 incher. i mean it all comes down to personal preference at the end of the day but if you're not experiencing significant hair loss / balding patterns and you're 15-18 i'd just stick to ru-58841 or KX-826

yes i agree with that, why would you still promote fin over dutasteride?

 

[Syna]

yes, but if you're in that age range of 15-18 and on AAS, i'd recommend either hopping on a topical hair prevention rather then going to DHT or FIN cause penile length / growth is law bro, can't have the foids with a 4 or 5 incher. i mean it all comes down to personal preference at the end of the day but if you're not experiencing significant hair loss / balding patterns and you're 15-18 i'd just stick to ru-58841 or KX-826

just go for KX-826, mogs RU brutally, higher binding affinity, no observed systemic re absorption, cleaner mechanism, aced phase 3 trials.

 

[looksmin]

just go for KX-826, mogs RU brutally, higher binding affinity, no observed systemic re absorption, cleaner mechanism, aced phase 3 trials.

ngl i don't know anything about KX-826 yet. gonna look up on that but tbh i prolly wont need hair prevention, i have really good hair genetics in my family. my dad who is 66 has a full head of hair with no balding or thinning at his old ass age jfl. ill get one just to have it on standby once i hop on more AAS

 

[looksmin]

yes i agree with that, why would you still promote fin over dutasteride?

well like as I said 1mg of fin daily would be enough for MOST men who are showing signs of androgenic alopecia. it would significantly slow down and or stop hair loss from androgenic alopecia. the argument can be made for some guys that have an aggressive form of androgenic alopecia that started early on but even for those guys 1mg of fin might be enough. BUT if they're past like 22 years old then I mean why not hop on DUT i guess, DHT doesnt have any good effects on you once you end puberty so might as well nuke it.

 

[Syna]

ngl i don't know anything about KX-826 yet. gonna look up on that but tbh i prolly wont need hair prevention, i have really good hair genetics in my family. my dad who is 66 has a full head of hair with no balding or thinning at his old ass age jfl. ill get one just to have it on standby once i hop on more AAS

Yeah, tho assuming the use of androgens like the ones you promote like primo, tren, test, mast and all of them would have a negligible effect on your hair is dumb, go for hair prevention, you're also preventing androgens from raping your collagen and elasticity, there's literally no downsides to it.

 

[looksmin]

Yeah, tho assuming the use of androgens like the ones you promote like primo, tren, test, mast and all of them would have a negligible effect on your hair is dumb, go for hair prevention, you're also preventing androgens from raping your collagen and elasticity, there's literally no downsides to it.

I personally don't think i'd hop on primo or mast, my upcoming cycle i plan on running test, var, hgh, and maybe microdosed tren and some other compounds. not too sure yet, still in the looks and researching era of what i am looking for and planning on to add into the cycle.

 

[Syna]

well like as I said 1mg of fin daily would be enough for MOST men who are showing signs of androgenic alopecia. it would significantly slow down and or stop hair loss from androgenic alopecia. the argument can be made for some guys that have an aggressive form of androgenic alopecia that started early on but even for those guys 1mg of fin might be enough. BUT if they're past like 22 years old then I mean why not hop on DUT i guess, DHT doesnt have any good effects on you once you end puberty so might as well nuke it.

above 18, the 20s range is a meme, finasteride is pathetic compared to dutasteride, again, nuking it all has zero downsides, also finasteride is quite pathetic at blocking the DHT conversion from AAS like test and ment that can convert to DHT, specially at supraphysiological doses, no point using fin when you have such a superior alternative with negligible downsides and ideal for hair loss prevention and anti aging. TLDR use dutasteride.

 

[Syna]

I personally don't think i'd hop on primo or mast, my upcoming cycle i plan on running test, var, hgh, and maybe microdosed tren and some other compounds. not too sure yet, still in the looks and researching era of what i am looking for and planning on to add into the cycle.

Then why do you promote them for E2 control? also why the EQ propaganda?

 

[looksmin]

Then why do you promote them for E2 control? also why the EQ propaganda?

well I promote them because they aren't horrible compounds. the benefits are discussed in the guide I posted. I just don't think the compounds meet my needs. my frame is already good before hopping on AAS, I have a shoulder dominant physique, wide clavicles and a semi narrow waist.

 

[looksmin]

at the end of the day everyone has different goals, if the compound aligns with your goals take it.

 

[MedSlayer]

penile development stops at around age 16-18, DHT serves no other purpose but penile enlargement, after 18 you don't need it, there's no good argument for not using dutasteride, DHT rapes your hair follicles and your skin elasticity, specially when you are using supraphysiological amounts of androgens in this case test.

DHT is still important in adulthood just not in excess or in certain area like the hairline

 

[Syna]

well I promote them because they aren't horrible compounds. the benefits are discussed in the guide I posted. I just don't think the compounds meet my needs. my frame is already good before hopping on AAS, I have a shoulder dominant physique, wide clavicles and a semi narrow waist.

none are worth the collagen and hair rape when you can use a conventional AI for that purpose, you never answered why do you like EQ but okay.

 

[Syna]

DHT is still important in adulthood just not in excess or in certain area like the hairline

like for?

 

[looksmin]

none are worth the collagen and hair rape when you can use a conventional AI for that purpose, you never answered why do you like EQ but okay.

if you lose collagen and hair rape just use fin, dut or a topical prevention serum and for collagen just use a retinoid?

i don't think I have to say why I like eq lol just personal preference when paired with mast

 

[Syna]

if you lose collagen and hair rape just use fin, dut or a topical prevention serum and for collagen just use a retinoid?

i don't think I have to say why I like eq lol just personal preference when paired with mast

fin and dutasteride ain't doing shit, using a topical 5ar blocker still doesn't avoid the collagen rape fully, same with a retinoid, the ROI is pretty dogshit, if you are gonna use a compound as collagen and hair raping as primo and mast you are better off using low dose tren from an ROI view point, you will get way way way bigger and leaner and more shredded than with primo or mast.


Regarding EQ, shitty ass ROI as well, not to mention, but there is no correct ratio for EQ the 1:1 or 1:2 is just guessing at its finest, EQ's effect on estrogen is weird af and super unpredictable, you cant just predict what the ideal ratio for someone, it doesn't even work like a regular Al cause it doesn't bind and block the aromatase enzyme, it rather competes with test for the aromatase enzyme and shifts what gets produced, which is more estrone and less bioidentical E2, all this without even mentioning that it takes a lot of time to reach serum concertation levels. And yes, the actual E2 can still tank, all this nonsense is because Cheap ECLIA tests cross-react with EQ metabolites and E1, so they can register or read a high E2 when real LC-MS/MS shows it crushed.

Because EQ uses a undecylenate ester it takes a lot of time to exit your system, so again, there is not correct ratio for EQ, so if the 1:1 or 1:2 crashes your estrogen it's going to take a lot of time to recover and re adjust, also the neuropsychiatric effects of EQ on the CNS are absolute dogshit, also EQ is the AAS that raises the most and the fastest your hematocrit and RBC, the appetite stimulation is also a meme than gets mogged by GHRP 2-6 or by a ghrelin mimetic and agonist like MK677, so i look at it and i see no advantage to using EQ over a conventional AI.

 

[MedSlayer]

like for?

DHT convert into important neurosteroids
Primary driver of facial hair growth
Primary driver of erection and libido

 

[Syna]

DHT convert into important neurosteroids
Primary driver of facial hair growth
Primary of erection and libido

It's a meme, the ed is a nocebo effect, already been debunked to death, DHT plays a key role for facial hair growth but it's not as relevant as genetics, would much rather have intact collagen and a head full of hair than a beard.

 

[Syna]

DHT convert into important neurosteroids
Primary driver of facial hair growth
Primary driver of erection and libido

 

[makeaway]

It's a meme, the ed is a nocebo effect, already been debunked to death, DHT plays a key role for facial hair growth but it's not as relevant as genetics, would much rather have intact collagen and a head full of hair than a beard.

syna i love you i hate you i fucking love you

 

[MedSlayer]

View attachment 50322

It's a meme, the ed is a nocebo effect, already been debunked to death, DHT plays a key role for facial hair growth but it's not as relevant as genetics, would much rather have intact collagen and a head full of hair than a beard.

Ill have to read the studies deeper
DHT only cause fibrosis on your scalp not skin
DHT only affects elastin not collagen
DHT is still important because of its metabolites who are neurosteroids involved in GABA

 

[Syna]

Ill have to read the studies deeper
DHT only cause fibrosis on your scalp not skin
DHT only affects elastin not collagen
DHT is still important because of its metabolites who are neurosteroids involved in GABA

it affects both collagen and elastin, dutasteride doesn't cause a significant suppression relevant enough to not use a 5ar blocker, if anything test and E2 increase after using 5ar blockers, and E2 plays a way more relevant role from a neuroprotective standpoint than DHT, It regulates synaptic plasticity, protects neurons from oxidative stress and apoptosis, and modulates mood, cognition, and memory, and if the neurosteroid effect was relevant enough it would've been noticed in the studies as a possible contraindicator against prescribing a 5ar blocker to patients with prostate enlargement and androgenic alopecia.

 

[Syna]

Ill have to read the studies deeper
DHT only cause fibrosis on your scalp not skin
DHT only affects elastin not collagen
DHT is still important because of its metabolites who are neurosteroids involved in GABA

It does affect both elastin and collagen
When DHT binds to an AR inside a fibroblast it alters the genetic transcription factors of that cell, altering the production of collagen and elastin at the same time from the whole cell, also, when androgens trigger this receptors, they activate the MAPK/ERK pathways which induce the downregulation of elastin and collagen synthesis when they bind to the AR(because the androgens signal the cells to lower the production of pro collagen and tropoelastin which are one of the most essential elements for your skin elasticity and quality), and also trigger the upregulation of degradation (MMPs) (because androgens upregulate the production of factors like that "cut" MMP-1 cuts collagen, while MMP-12 cuts elastin.

 

[MedSlayer]

it affects both collagen and elastin, dutasteride doesn't cause a significant suppression relevant enough to not use a 5ar blocker, if anything test and E2 increase after using 5ar blockers, and E2 plays a way more relevant role from a neuroprotective standpoint than DHT, It regulates synaptic plasticity, protects neurons from oxidative stress and apoptosis, and modulates mood, cognition, and memory, and if the neurosteroid effect was relevant enough it would've been noticed in the studies as a possible contraindicator against prescribing a 5ar blocker to patients with prostate enlargement and androgenic alopecia.

The reason early studies from the 90s and 2000s didn't list neurosteroid depletion as a strict contraindication is because those original trials focused almost entirely on pelvic mechanics, completely ignoring the brain's 5AR pathways. While the brain is highly adaptable , which is why the average man adapts to the increased T and E2 and feels fine if you are genetically predisposed, it will make you feel like shit. That is exactly why Health Canada now labels it as a potential side effect.

 

[MedSlayer]

It does affect both elastin and collagen
When DHT binds to an AR inside a fibroblast it alters the genetic transcription factors of that cell, altering the production of collagen and elastin at the same time from the whole cell, also, when androgens trigger this receptors, they activate the MAPK/ERK pathways which induce the downregulation of elastin and collagen synthesis when they bind to the AR(because the androgens signal the cells to lower the production of pro collagen and tropoelastin which are one of the most essential elements for your skin elasticity and quality), and also trigger the upregulation of degradation (MMPs) (because androgens upregulate the production of factors like that "cut" MMP-1 cuts collagen, while MMP-12 cuts elastin.

Androgens doesnt trigger MAPK/ERK enough for it to matter unless its in the scalp and you have male pattern baldness
Same goes for MMPs

 

[Syna]

The reason early studies from the 90s and 2000s didn't list neurosteroid depletion as a strict contraindication is because those original trials focused almost entirely on pelvic mechanics, completely ignoring the brain's 5AR pathways. While the brain is highly adaptable , which is why the average man adapts to the increased T and E2 and feels fine if you are genetically predisposed, it will make you feel like shit. That is exactly why Health Canada now labels it as a potential side effect.

I was not aware of that, good to know, i still think that if the neurosteroid depletion was as relevant many people would report a lot more side effects related to depression and other mood swings, such a case i think dutasteride should be administered as tolerated if that was to happen.

 

[MedSlayer]

I was not aware of that, good to know, i still think that if the neurosteroid depletion was as relevant many people would report a lot more side effects related to depression and other mood swings, such a case i think dutasteride should be administered as tolerated if that was to happen.

+ you still need DHT for that mogger stubble and slay all the estrogen and fertility maxxed foid in their 20s

 

[Syna]

Androgens doesnt trigger MAPK/ERK enough for it to matter unless its in the scalp and you have male pattern baldness

They do, hence why a combination of heavy androgen load and oxidative stress is what ages body builders so much, dermal fibroblasts are found everywhere in human skin, not just the scalp. Also receptor density and receptor function are not the same, humans only have one single gene for the Androgen Receptor, and the AR protein in a facial fibroblast is biochemically identical to one in the scalp, so they do trigger the MAPK/ERK the same way, at least from the point DHT becomes relevant when altering the functions already discussed and therefore affecting the skin quality.

https://www.mdpi.com/2079-9284/12/5/207 :

 

[Syna]

They do, hence why a combination of heavy androgen load and oxidative stress is what ages body builders so much, dermal fibroblasts are found everywhere in human skin, not just the scalp. Also receptor density and receptor function are not the same, humans only have one single gene for the Androgen Receptor, and the AR protein in a facial fibroblast is biochemically identical to one in the scalp, so they do trigger the MAPK/ERK the same way, at least from the point DHT becomes relevant when altering the functions already discussed and therefore affecting the skin quality.

https://www.mdpi.com/2079-9284/12/5/207 :
View attachment 50330

I also need to mention there are studies on 5ARI siblings that show a much higher skin quality compared to the sibling with normal DHT.

 

[MedSlayer]

They do, hence why a combination of heavy androgen load and oxidative stress is what ages body builders so much, dermal fibroblasts are found everywhere in human skin, not just the scalp. Also receptor density and receptor function are not the same, humans only have one single gene for the Androgen Receptor, and the AR protein in a facial fibroblast is biochemically identical to one in the scalp, so they do trigger the MAPK/ERK the same way, at least from the point DHT becomes relevant when altering the functions already discussed and therefore affecting the skin quality.

https://www.mdpi.com/2079-9284/12/5/207 :
View attachment 50330

1.Identical receptors behave differently depending on the environnement
2.Bodybuilders age asf because they live under extreme stress and take a huge amount of androgens which do not apply to the average joe

 

[MedSlayer]

I also need to mention there are studies on 5ARI siblings that show a much higher skin quality compared to the sibling with normal DHT.

will look into it later but rn i have to study see ya later

 

[Syna]

1.Identical receptors behave differently depending on the environnement
2.Bodybuilders age asf because they live under extreme stress and take a huge amount of androgens which do not apply to the average joe

but we were discussing the use of dutasteride in the context of PED usage lol, even if not experienced with the same extent the average joe still experiences the same effect by androgens, not as insanely brutal but it's there.

The receptor thing is a buzzword in this context, cell types can differ dermal fibroblast on the face and a dermal fibroblast on the body share the exact same cellular environment and genomic DNA, 5ARD siblings literally showed meaningful differences regarding skin quality when scanned on the DHT deficient sibling, the only difference noticed was a micro penis obviously, (super off topic but this is one of the strongest proofs about DHT NOT growing bones cause no skeletal deficiency was noticed).

I still think the neuro steroid depletion is really not that significant or relevant to be a point against the use of dutasteride in my opinion, also the raise of E2 should still compensate to some degree, in the weird case any side effect by dutasteride is present it can be mitigated super easily spacing the doses, though it should not apply for the vast majority of the population as E2 is way more relevant from a neuroprotection standpoint than DHT so the compensation of E2 should fix any issues, even if something weird occurs, again, can be easily mitigated spacing the doses.

 

[surgerymax]

well like as I said 1mg of fin daily would be enough for MOST men who are showing signs of androgenic alopecia. it would significantly slow down and or stop hair loss from androgenic alopecia. the argument can be made for some guys that have an aggressive form of androgenic alopecia that started early on but even for those guys 1mg of fin might be enough. BUT if they're past like 22 years old then I mean why not hop on DUT i guess, DHT doesnt have any good effects on you once you end puberty so might as well nuke it.

untrue especially on a test cycle when u have multiple times as much DHT , you need at least 2.5mg dut let alone 1mg fin being enough